This article followed up on the working memory deficits seen in a well established Rat Schizophrenia model, by trying to replicate the same in Rhesus monkeys. They produced ventral hippocampal lesions during the infancy of Rhesus Macaques, using multiple small doses of Ibotenic acid. They speculated that this would result in Prefrontal cortical maldevelopment and finally working memory deficits in adult state. They assessed two major components of Working memory, namely monitoring and maintenance of information. The former was assessed using object self-ordered (Obj-SO) [mediated by the dorsolateral prefrontal cortex], while the latter using session-unique delayed nonmatch-to-sample (SU-DNMS) task [mediated by the ventral lateral prefrontal cortex].